Cart
sales@molnova.com
12-Hydroxydodecanoic acid
CAS No. 505-95-3
12-Hydroxydodecanoic acid( 12-hydroxylauric acid )
Catalog No. M19613 CAS No. 505-95-3
12-hydroxydodecanoic acid is the substrate of the human glutathione-dependent formaldehyde dehydrogenase.
Purity : >98% (HPLC)
COA
Datasheet
HNMR
HPLC
MSDS
Handing Instructions
| Size | Price / USD | Stock | Quantity |
| 50MG | 35 | In Stock |
|
| 100MG | 50 | In Stock |
|
| 200MG | 72 | In Stock |
|
| 500MG | 125 | In Stock |
|
| 1G | Get Quote | In Stock |
|
Biological Information
-
Product Name12-Hydroxydodecanoic acid
-
NoteResearch use only, not for human use.
-
Brief Description12-hydroxydodecanoic acid is the substrate of the human glutathione-dependent formaldehyde dehydrogenase.
-
Description12-hydroxydodecanoic acid is the substrate of the human glutathione-dependent formaldehyde dehydrogenase.
-
In Vitro——
-
In Vivo——
-
Synonyms12-hydroxylauric acid
-
PathwayOthers
-
TargetOther Targets
-
RecptorOthers
-
Research Area——
-
Indication——
Chemical Information
-
CAS Number505-95-3
-
Formula Weight216.32
-
Molecular FormulaC12H24O3
-
Purity>98% (HPLC)
-
SolubilityDMSO:10 mM
-
SMILESOCCCCCCCCCCCC(O)=O
-
Chemical Name——
Shipping & Storage Information
-
Storage(-20℃)
-
ShippingWith Ice Pack
-
Stability≥ 2 years
Reference
1.Sanghani PC et al. Human glutathione-dependent formaldehyde dehydrogenase. Structures of apo binary and inhibitory ternary complexes. Biochemistry. 2002 Sep 3;41(35):10778-86.
molnova catalog
related products
-
S961 TFA (1083433-49...
S961 TFA is A high affinity insulin receptor (IR) antagonist with IC50 for hir-a, hir-b and human insulin-like growth factor I receptor (higf-ir) at 0.048, 0.027 and 630 nM, respectively.
-
PACAP 1-38
PACAP 1-38 is a neuropeptide with 38 amino acid residues.?
-
trans-2-Butene-14-di...
3-Hexenedioic acid is a normal human unsaturated dicarboxylic acid metabolite with increased excretion in patients with Dicarboxylic aciduria caused by fatty acid metabolism disorders The urinary excretion of 3-Hexenedioic acid is increased in conditions of augmented mobilization of fatty acids or inhibited fatty acid oxidation.
